[alex]

– keep E2 as high as possible (without sideeffects)

– cardio

– diet (high monounsaturated fats) / avoid excess bodyfat

– low dose statin (like 5mg rosuvastatin 1-2x/week)

[alex]

drink 20g Glutamine + curcumin (like Cure-Coming) with 1L water in the morning right after waking up and skip meal6 – try to give your digestion a break over the night

[alex]

Get some 1ml zero dead volume syringes,

get some 30G needles.

Grap some belly fat/skin,

inject it there. (or wherever you want..)

[alex]

Still cant see a picture bud – Can you copy and paste it in here?

https://imgbox.com/sEw22XDK

Gonna try it like this if its allowed not sure how otherwise

[/quote]
he just postet the link there😋
everything fine with website version.. (app I dont see anything too)

[alex]

I dont see estrogen? Would be good to know, due to the low HDL, maybe estrogen is quite low – then you could just remove the primo for some weeks and see where u at. Also donate some blood and/or increase telmisartan to 40mg and it looks like you could handle more carbs, good hba1c.

How do you feel? Water retention, libido, achy joints (or vice versa) ?

[alex]

depending on your lipds..

I prefer 5mg rosuvastatin once or twice weekly over any supplement

[alex]

dr. scott stevenson says it, so be it!

[alex]

Angiotensin II seem to have the effect to keep unusual high hematocrit stable over long periods of time even if not needed.. ARB like telmisartan just has some regulating effect which leads to normalized hematocrit over time.. dont know why, dont know how long it takes, definitely does not makes blood thinner.

John Jewett said he had not to donate blood for months while on telmi..
My hematocrit is high normal and didnt change a bit after one year of 40mg telmi🤷🏻‍♂️

effect probably quite low

[alex]

yeah the description rebound makes not really sense here – before a rebound could occur, the enzymes had to upregulate which then would lead to permanently increase of AI usage, which is not the case..
women run same dose of AIs over years with almost no adjustments..
you can have rebounds from drugs, for example abusing opiates, receptors upregulate, need more opiates for same effect. then stop to zero, receptors still in an upregulated state, rebound occurs with all the „rebound“symptoms (=withdrawl).. receptors and agonists changed stage of homeostasis..

so .. due to the longer half life of adex and letro, and maybe even longer than rebuilding new enzymes after exemestane(for men I read couple times something about 6-12h) – the here mentioned „rebound“effect would be lower with adex and letro instead of exe..!?

[alex]

I ordered every day with careem or some arab uber lookaliketaxi to my condo. Groceries, pharmaceuticals, clothes (which the driver waited until I tried them!)
The most expensive and suspicious part is trying to buy simple whey protein..
Daily food service, they just ask what you need. same with pharma.
OK – probably not 100% legal.😂
Kinda same in thailand, turkey or greece and still if you ask straight for what you want, you get it even in official hospital pharmacies. In this countries, laws are not all the same, „a law is a law“ as western countries define it, is just not the same understanding there.

except narcotics, there is definitely the line..

[alex]

just buy your peds there in the pharmacies.
legal
original
cheap

..only nanny-states care about too muscular citizens 😂

[alex]

that doesn’t make sense to me, are we taking about the same? maybe I dont get it 🙁

I think the reason using dhts like mast to control estrogen instead of using AIs is to keep your E2 as high as possible for the health benefits, without the nasty sideeffects like gyno for example.
I understood this as shifting the androgen:estrogen ratio towards androgens, which results in less E2 sides..? (due to exert opposing effects in several tissues, some receptor up/down regulations, binding competition and so on and so on..)

if its just about reducing E2 in the bloodwork, where is the difference to just using an AI?

The first 2 years using test I went to the lab almost every second month checking serum estradiol E2 (refrange 41-159 pmol/L, switzerland, method of measurement dont know). mostly just because of gyno paranoia…

so I testet various testo-only doses; various tests with testo-exemestan/ari/letro. (here the E2 values from the bloodwork were always kinda coherent for me)

testo-proviron (here shbg very low, free test high, E2 higher than with same test-only dose, but reduced E2 sides with provi!).
Testet couple times test with mast and lately primo.. (trtplus, here only primo lowers my E2 on the bloodresults, even so I feel less estrogrenic with mast, but 250test with 100mast I see no difference at my serum E2)

for example,
old bloodwork here 250test cyp e7d, no ai no dht, E2 179pmol/L. my libido was gone, nipple hurt, moonface, I almost cried watching random netflix series..
There I started using AIs, always kept E2 somewhere in range but below 100pmol, even with higher doses test. (yes today I probably would at first inject more frequently 😅)
felt very good but my lipds got worse over time.

later, shifted towards „safer use model“ no AI but dhts:
bloodwork 250test with 100mast e3d, E2 310pmol/L! but I felt just fine. no water, horny af, no weird sensations on my nipples from just wearing a shirt, and so on.
I tried 250test with 200mast e3d too, I think E2 went only a bit down around ~250ish(but had lower bodyfat at this time..) but my joints started to hurt and I felt like crap.. (again maybe due to the lower bodyfat, not sure if conclusions are valid..)

I have various bloodworks here like this.
Only thing on paper where I see valid differences on high/low E2 is hdl/ldl.
The higher my serum E2, the better.(especially hdl )
But just from lookin at E2 I dont see any useful data for me..

Do you see why I can’t understand the focus on E2 in bloodwork for adjusting cycles while using dht?
Is there some different E2 measuring in UK (or switzerland)..!?
Or are we talking about different things?

[alex]

hmm mast does only slightly reduce e2 imo.

the anti-estrogenic effect is more „serm-like“ (or lets say it kinda competes with e2 about some receptors, but instead of activating them it just binds and blocks, so E2 loss its effects in some tissues..)

I see a more E2 lowering effect on my bloodwork from primo, even so mast is still stronger in masking e2 sideeffects for me..

Proviron works well too for me masking e2 sideeffects, but with proviron my E2 is actually higher than without.
guess its due to the very low shbg on provi..

so.. I read many times here to check bloods for e2 when using dhts instead of an ai to control e2, but I dont see what the e2 marker will tell me then due to the different mechanisms of controlling e2 with dhts..

how can I interpret the bloods ?
personally I just check how I feel on different test:dht ratios.. but is there a way to valid interpret bloods ?

[alex]

anecdotally, abusing gh makes it worse.
and there are some rare case studies of treatment with hyaluronidase injections

[alex]

dont try to do pct and get in shape at the same time, so be in shape before.

for restarting hpta, a slight caloric surplus is preferable.